Your position on priated movies Essay Example | Topics and Well Written Essays - 750 words - 1

Your position on priated movies - Essay Example Pirated movies might be great for some consumers, but they have a negative impact on the film industry, can influence consumers to pay good money for horrible quality, and is considered a form of theft. Since pirated movies are stolen movies, those who create and distribute pirated movies should be treated and punished as criminals. One of the greatest misconceptions of movie piracy is that wealthy filmmakers cannot possibly be affected by losing sales on their films. Indeed, many people who engage in film piracy condone their actions by pointing out that members of film crews make more money than is necessary and can afford to allow a few movies to be released without requiring pay (Aicher 61). What they do not realize is that the profits made from films do not simply line the pockets of the filmmakers, but are also used for future film productions. This negative financial impact on the film industry will result in fewer high budget films, including many of the popular franchises, such as Marvel’s The Avengers, Star Trek, and The Hunger Games, and a significant decrease of releases in general. Furthermore, movie theaters throughout the world would feel the repercussions of film piracy if the film industry ceased to release movies to be shown at the theaters. Theaters would then lose sales, make no return profit, and be forced to shut down business. Though it may be unfathomable, film piracy has the ability to completely destroy the renowned success of the world’s collective film industry (Segrave 115). Regardless of whether or not people think filmmakers make too much money, it needs to be known that for any industry, an individual person or any entire company requires payment to continue doing what they do best. If those heavily engaged in film piracy persist on taking and selling product that are not their own, they will not only rob the film industry of its legacy but destroy entertainment for people worldwide. The filmmakers are not the only

Explain the significance of property in Lockes theory of natural Essay

Explain the significance of property in Lockes theory of natural rights - Essay Example Locke states specifically that â€Å"mens lives, liberties and estates, I call by the general name property†3. So here, it is apparent that Locke defines property in a way that is quite unique and distinct from the mainstream definition we are accustomed to. Again, Locke states that â€Å"by property, I must be understood here as in other places to mean that property which men have in their persons as well as goods†4. Locke repeats the wider definition of property which seem to define property in relation to the estates and goods a person owns as well as a persons life as a living person. Looking at Lockes definition of property in the macrocosmic sense, it is apparent that he was arguing that once a person has a life, he has a property. This property is of a value even if the person does not own any property. Locke puts forward several cases and arguments that supports his points in England of his era. Most of these arguments are rooted in Biblical principles and precepts and it weaves into the wider society by building a hedge around the human lives to ensure that people get good and fair treatment wherever they find themselves. Locke argues that human beings have been given rights by God to take dominion over the earth and exercise control over the natural environment and the lesser creatures5. This is supported by the Bible in Genesis. Locke therefore states that â€Å"man should live and abide for some time upon the face of the earth†6. This therefore means that every individual on the earth has some rights to control some portion of the earth because it was an in inherent and inalienable right given to Adam by God. This therefore means that every human being is by extension, a representative of God who seeks to honour the purpose for which Adam was created: to take dominion and guard over the natural environment. Although God gave dominion to Adam, this was held exclusively by all human beings

Bacteria menningitis haemophilus influenza Essay

Bacteria menningitis haemophilus influenza - Essay Example It is responsible for lower respiratory tract infections in children. They are opportunistic pathogens and only become pronounced on exposure of the body to viral infections, immune dysfunction, and chronic inflammation. They stick to the cells by use of adhesions such as TAA. It causes pneumonia, bacteremia, and acute bacterial meningitis. It also causes infectious arthritis, osteomyelitis, epiglottitis, and cellulitis. Vaccination is available against the bacteria using their conjugates. However, non-encapsulated strains do not get affected and cause otitis media (ear infection) and conjunctivitis. Causative Agent The Haemophilus influenza meningitis is caused by Haemophilus influenza type b bacteria. The bacteria are in the kingdom of bacteria; phylum is proteobacteria; the class is gamma proteobacteria; the order is pasteurellales; the family is pasteurellaceae; the genus is haemophilus and the species is Haemophilus influenza. It is a rod shaped gram-negative bacteria coccobacil lus bacterium (Evans and Brachman 14). It possesses the aerobic nature but can also grow as a facultative anaerobe. It has two serotypes; the encapsulated and the non-encapsulated strains. The encapsulated strains were classified into H. Influenza: a (Hia), b (Hib), c (Hic), d (Hid), e (Hie), and f (Hif). This classification is based on the capsular antigens present. The non-encapsulated are non-typable due to the lack of the capsular serotype. They produce inflammatory responses and remain less invasive. The inflammation of the meningeal membranes, which covers the brain, causes the symptoms of meningitis. This makes them resistant to the H. influenza vaccine that is induced to vaccinate against the bacteria. This leads to conditions like pneumonia and otitis media often seen in children, which are the complications, associated with meningitis. H. influenzae, in a Gram stain of a sputum sample, appear as Gram-negative coccobacilli H. Influenzae on an agar plate. Pathogenesis The pa thogenesis of the bacteria is not well defined. However, the main factor to its pathogenesis is the type b polysaccharide capsule. This capsule harbors the antigen responsible for the effects associated with the disease. The encapsulated bacteria can penetrate the surfaces of the epithelium and get into the blood capillaries without hindrance. The antigens present on the capsule allow for its direct penetration. The capsule enclosure makes the bacteria resist phagocytosis by the phagocytes and, thus, its infiltration into the inside. The complement cascade of the host immune system cannot act on the bacteria, thus, making it get invasive. The non-encapsulated strain is lowly invasive compared to the encapsulated. This is because they lack the capsule, hence, the lack of antigens responsible for infiltrating the bilayer. However, they can induce the inflammatory response of different cells that later cause disease. This later leads to the symptoms associated with the disease. It is a lso abundantly evident after an infection of the upper respiratory region. The infection multiplies by spreading from the respiratory tract and then to the bloodstream and later spreads to the meninges. At the meninges, the pathogen induces inflammation and the complement activation. This inflammation around the meninges causes the meningitis associated with the disease. This affects nerve impulse transmission and, thus, causes illness or later death. The bacterium also produces beta lactamases, to aid in its resistance

Explain the significance of property in Lockes theory of natural Essay

Explain the significance of property in Lockes theory of natural rights - Essay Example Locke states specifically that â€Å"mens lives, liberties and estates, I call by the general name property†3. So here, it is apparent that Locke defines property in a way that is quite unique and distinct from the mainstream definition we are accustomed to. Again, Locke states that â€Å"by property, I must be understood here as in other places to mean that property which men have in their persons as well as goods†4. Locke repeats the wider definition of property which seem to define property in relation to the estates and goods a person owns as well as a persons life as a living person. Looking at Lockes definition of property in the macrocosmic sense, it is apparent that he was arguing that once a person has a life, he has a property. This property is of a value even if the person does not own any property. Locke puts forward several cases and arguments that supports his points in England of his era. Most of these arguments are rooted in Biblical principles and precepts and it weaves into the wider society by building a hedge around the human lives to ensure that people get good and fair treatment wherever they find themselves. Locke argues that human beings have been given rights by God to take dominion over the earth and exercise control over the natural environment and the lesser creatures5. This is supported by the Bible in Genesis. Locke therefore states that â€Å"man should live and abide for some time upon the face of the earth†6. This therefore means that every individual on the earth has some rights to control some portion of the earth because it was an in inherent and inalienable right given to Adam by God. This therefore means that every human being is by extension, a representative of God who seeks to honour the purpose for which Adam was created: to take dominion and guard over the natural environment. Although God gave dominion to Adam, this was held exclusively by all human beings

Social Factors in Language Learning Essay Example | Topics and Well Written Essays - 1250 words

Social Factors in Language Learning - Essay Example This paper declares that nowadays the English language undergoes several variations due to different factors. Among them people can distinguish regional, social, personal and even gender variations. The most widespread and obvious one is regional variation of the English language. This discussion explores that thanks to the popularity of American media and British culture the speakers of both models adopt certain vocabulary and pronunciation from each other. This process is commonly called accommodation. Apart from two main models of English regional variety of this language includes several dialectal variations. There are regional dialects of English both, in the USA and Great Britain. Dialects vary mostly in vocabulary and these differences are quite large and numerous. Unfortunately, this contrast between speakers sometimes leads to conflicts and bulling because one dialect is considered to be more privileged than the other. However, people should abandon this prejudice and treat everyone equally. Depending on people’s social identities, they use different types of language. There are many factors which predetermine the usage of certain constructions, words and intonation. They include the level of education, sex, occupying job and age. In some count ries with English speaking population social variation of the language does not play a huge role.

Smoking in communal places needs to be prohibited Essay Example for Free

Smoking in communal places needs to be prohibited Essay Smoking in communal places needs to be prohibited. All of us have heard about the secondhand smoke’s negative paraphernalia to human health, newly done investigation is usually dropping lighting about just how effective it actually is. A current analysis through the United National Institute on Drug Abuse (NIDA) observed that will secondhand smoke causes it to be more difficult with for smokers to give up smoking (Marzilli 2008). Analysts point out that secondhand smoke sets off nicotine cravings  which can adjusts brain purpose, compared to truly smoking cigarettes a smoke. In the event secondhand smoke possesses a real effective effect on smokers, and then what exactly should it complete for you to those that aren’t possibly lighting up? Secondhand smoke poisons others, in particular kids as well as pregnant women, at risk of breathing in cigarettes’ smoke. Furthermore, smoking in communal places endangers individuals who have the respiratory system health disorders. Tobacco contains the Carbon (II) oxide, which if inhaled will lead to the reduction of the carrying capacity of oxygen in the blood (Buddelmeyer Wilkins 2010). Carcinogenic compounds which is also present in tobacco is said by research to cause mutation of the DNA to any individual who inhales it and as a result lead infection cancer (Buddelmeyer Wilkins 2010). In a matter of fact, smoke from a burning cigarette ingests most to a non-smoker and does not get sucked in by the smoker’s lungs furthermore he/she escalates it to the air. The latest scientific tests display that nearly 69.79% of individuals ingesting cast-off smoke from communal smokers have a larger chance of getting respiratory system troubles when compared with smokers themse lves (Qwins 2013). Tens of thousands deaths per year are as a result of the effects of secondhand smoke of which are categorized into two thus; lung cancer and heart disease. Prohibiting smoking in public regions is often useful in several ways since it eradicates an act involving difficulties to acquitted individuals. In the event of prohibiting smoking of cigarettes in communal places, we lower or even totally remove the chance of placing non-smokers susceptible and or increasing the smokers’ chances of creating lung and heart glitches to themselves. Additionally, when many of us prohibit smoking cigarettes in communal places, many of us also cease the smokers from additionally increasing their likelihood of getting medical problems pertaining to themselves. Research clearly points out that prohibiting smoking in public areas saves more than 15 million heart attacks and 20 million lung cancer attacks a year nationwide (Qwins 2013). Though it is usually mentioned that not every individual who smoke in communal regions usually led to harming other people, It remains undeniable fact that cigarette smoking by itself is really a reason for health disorders. Its not ample to convey which the size of impacted individuals is relatively just a small fraction; profuse or perhaps few of, an individual put in danger is usually ample. It can be just the situation with cigarette smoking in communal places, therefore, needs to be restricted. References Buddelmeyer, Hielke, and Roger Wilkins. The effects of smoking ban regulations on individual smoking rates. Parkville] Vic.: Melbourne Institute of Applied Economic and Social Research, 2010. Print Marzilli, Alan. Smoking Bans. Point Counterpoint. New York: Kate Bothers, 2008. Print. Qwins, Leslie. Tobacco smoking. Lyon?: World Health Organization, International Agency for Research on Cancer, 2013. Print. Source document

Permeability of Plasma Membranes

Permeability of Plasma Membranes Introduction Plasma membranes are bi-layered membranes made up of amphiphillic molecules (having charged polar heads tending to be hydrophillic and uncharged fatty acid tails tending to be hydrophobic) that selectively allow entrance of certain large molecules into the cells cytosol and through which water and small non-polar molecules may freely diffuse. This experiment seeks to understand limited aspects of the permeability of the plasma membrane using the Elodea leaf membrane as model organism. Some of the factors upon which permeability of the plasma membranes of biological organisms depend are differences in pH on opposite sides of the membrane, temperature, osmolarity, expression of certain membrane receptors and the concentration gradients of various molecules. This experiment is very limited in scope and seeks to answer only the question of what is the time dependence for permeability of glycerol through the cell membrane. Other experiments have answered many of our questions regarding this and have resulted in mathematical equations describing these results. This experiment will use one of the formula derived from these prior experiments, the Ether:Water partition coefficient for alcoholsiii as a means of hypothesizing what the outcome of this present experiment will be. I have hypothesized that within seconds of exposure to a 0.3M (molar) hyper-tonic solution of glycerol, dissolved in an isotonic deionized water (dH2O)/sucrose solution, the Elodea leaf will plasmolyze irreversibly-an assumption I believe is supported by the fact that glycerols ether:water partition coefficient is only 0.00066iii. Further support for this supposition is the fact that glycerol has a relatively bulky chemical structureviii-owing to its three large, highly polar hydroxyl groups-and a large molecular weight of 92.0938 grams per mole. Alternatively, it may be hypothesized that the glycerol-being an aliphatic alcohol (see diagram in section IV(i) infra) which, itself makes up a part of the plasma membranevi-will be capable of more easily diffusing across the plasma membrane as compared to the sucrose, which cannot diffuse across the membrane, in which case not only will there be no severe plasmolysis but there may, instead, be a build up of turgor pressure inside the cell due to the inward movement of the alcohol and its confinement in the central vacuole. Methods In order to discover what molar concentration of sucrose will be needed in an aqueous solution to create a solution that is isotonic to the leafs cytosol I shall perform a bifurcated experiment in which the first part shall be to determine this concentration. Part two of this experiment will be to determine the period of time it takes for glycerol to diffuse across the plasma membrane. In order to determine which molar solution of sucrose is isotonic to the cytosol of the Elodea cell I labeled 6 micro-centrifuge tubes with the markings: 0.2M, 0.3M, 0.4M, 0.5M, 0.6M and isotonic respectively and using an adjustable pipette placed 1000 ÃŽÂ ¼L of premixed sucrose solution of each of the indicated molarities into the respective tubes. In each of these tubes I placed an Elodea leaf and allowed them to sit for approximately five minutes [my observations of plasmolysis along with photographs of leaves in similar states to what I observed are provided in table 2 of the Table of observations of plasmolysis and photographs #2-#6 in the Photograph table which can be found in sections III(A) (B) respectively.] While awaiting the leaves to finish soaking I viewed a dry mounted Elodea leaf under a microsocpe using 20X and 40X objectives with 10X ocular so as to have a better idea of what a normal Elodea leaf looks like for comparison to the viewing of the wet mounts [photo of a leaf in similar state to what I observed is provided as photo #1 in the Photograph table of section III(B).] I then labeled 6 microscope slides using the same concentrations I used when labeling the micro-centrifuge tubes. After five minutes I prepared an individual wet mount of an Elodea leaf by placing a leaf from a micro-centrifuge tube onto a microscope slide, bearing its respective molarity, with the upper surface of the leaf face up. I placed a cover slip over the leaf and gently tapped the cover slip so as to seat it onto the slide and to remove any excess solution. I then viewed the wet mount-searching for indications of plasmolysis-under a microscope using the same 20X and 40X objective lenses and the 10X ocular lens I had viewed the dry mount and recorded my observations then repeated this process for each of the leaves in the remaining tubes. I was unable to obtain photos of my observations but I have included photos downloaded from the internet which were similar to what I had observed and provided them in tables 1-6 of section III(B). Having established which molarity of sucrose solution was isotonic with the cytosol of the cell (see table in section III(A)) I calculated the quantities of sucrose, glycerol (test solution) and 1-Propanol (counter test solution) I would need for the second part of this study. In those calculations I used the data presented in table 1 below. My calculations are presented in the Table of Calculations, table 3 of section III(C) infra. I plugged the results I obtained from table 3 into the formula C1 x V1 = C2 x V2 so that I may calculate the volumetric quantity of each of these chemicals I would need to add to each of my two 1 x 103 ÃŽÂ ¼L test solutions, my calculations for each may be found in Table 4 of section III(C). Using those calculation I then added the quantities of sucrose to each of the other two chemicals and subtracted the sum from the final volume of solution (1000 ÃŽÂ ¼L) I would be creating so that I will know the volume of deionized water (dH2O) I would need. Those calculations are shown in table 5 of section III(C). Using these calculations I then prepared 5 new micro-centrifuge tubes as follows: 3 tubes each containing a 1000 ÃŽÂ ¼L isotonic (0.4M) sucrose solution (one of which is to be used as a negative control); the fourth containing an aqueous solution of isotonic (0.4M) sucrose and 0.3M glycerol mixtures; and the fifth containing an aqueous solution of isotonic (0.4M) sucrose and 0.3M 1-Propanol mixtures (counter control). I placed one Elodea leaf into each of the 3 isotonic solutions and allowed them to soak for approximately five minutes. After five minutes I prepared a wet mount of the first of the 3 leaves as previously described. After viewing the first leaf (the negative control) I placed the second leaf on a slide and added 2 drops of the 0.3M glycerol/Sucrose solution to the slide then viewed and recorded my observations. I then prepared the third leaf using 2 drops of the 0.3M glycerol/Sucrose solution and viewed to be certain I obtained the same result as the last slide then after approximately 30 seconds added 2 drops of 1-Propanol/Sucrose solution (the counter test solution) to see if this would have an effect opposing that of the glycerol/Sucrose solution and recorded my observations which I describe next. Results A. Table of observations of Plasmolysis Table 2: Plasmolysis observations within five minutes of Elodeausing different sucrose solutions. Sucrose concentrations Plasmolysis observed (Y/N) Sucrose concentrations Plasmolysis observed (Y/N) B. Photograph tables (Photographs of Elodea leaves in various solutions): 1. Normal leaf (similar observation as prior to placing in solution) 2. Hypo-tonic solution (similar to observation as seen in 3. Isotonic solution (similar observation as in the isotonic solution and the ~0.4M-0.5M sucrose solutions) 4. Hyper-tonic solution (similar observation as seen in the 0.6M sucrose solution) 5. Plasmolysed leaf (similar observation as would have been seen in hyper-tonic solutions) 6. Plasmolysis Recovery (did not observe any recovery events but this is what I would also have been looking for had plasmolysis recovery taken place) C. Tables of Calculations: Table 3: Calculations for concentration of 0.3M glycerol/Sucrose solution. Amount of sucrose needed: [.137g] x V = 0.4M x .001 L V = (.0004g/L) / (0.137g) = 0.002919 L or 2.91 x 103mL Amount of glycerol needed: [0.028g] x V = 0.3M x .001 L V = (.0003g/L) / (0.028g) = 0.01071 L or 10.7 x 103mL Amount of 1-Propanol needed: [0.018g] x V = 0.3M x .001 L V = (.0003g/L) / (0.018g) = .01667 L or 16.7 x 103mL Table 4: Calculations of volumetric quantities of each chemical needed to make 1000 ÃŽÂ ¼L of each solution. 2.92 ÃŽÂ ¼L sucrose+ 10.7 ÃŽÂ ¼L glycerol + x(dH2O) = 1000 ÃŽÂ ¼L 13.62 ÃŽÂ ¼L + x(dH2O) = 1000 ÃŽÂ ¼L x(dH2O) = 1000 ÃŽÂ ¼L 13.62 ÃŽÂ ¼L x(dH2O) = 986.38 ÃŽÂ ¼L 2.92 ÃŽÂ ¼Lsucrose + 16.7 ÃŽÂ ¼L 1-Propanol + x(dH2O) = 1000 ÃŽÂ ¼L 19.62 ÃŽÂ ¼L + x(dH2O) = 1000 ÃŽÂ ¼L x(dH2O) = 1000 ÃŽÂ ¼L 19.62 ÃŽÂ ¼L x(dH2O) = 980.38 ÃŽÂ ¼L Table 5: Calculations of amounts to add to each solution. Discussion At first viewing I did not quite understand what was happening as I had not previously seen an Elodea leaf that presented without its large central vacuole let alone one that presented with chlorophyll throughout the entire cytosolic space. Having consulted with my fellow researchers (one of which obtained findings similar to mine in her experiment), none of whom had explanations for this result, I shall instead provide a summary of what I observed and what I had expected to observe. I had expected my first hypothesis to be borne out regarding the outward movement of water across the membrane and toward the hyper-tonic glycerol solution providing a sighting as in photograph 5, however what I discovered was an Elodea leaf showing absolutely no sign of plasmolysis. Instead of the expected I saw what was a leaf that appeared to be in a state of iso-osmolarity with its environment which would have been expected only in an isotonic solution as in photograph 3. There, also, was no turgor pressure as would have been seen in photograph 2 had the alternative hypothesis of inward movement of glycerol across the plasma membrane been borne out. Finally, had there been a plasmolysed cell the addition of the counter test solution of 1-propanol should have caused recovery as seen in photograph 6 but being I was unable to obtain a plasmolysed cell I was also unable to observe recovery of such cell. The results of this experiment has left me unable to either accept or reject either of the two hypotheses provided above.